MSRA

Aliases
  • EC 1.8.4.11
  • MSRA
  • PMSR
  • Peptide Met(O) reductase
  • Peptide-methionine (S)-S-oxide reductase
  • Protein-methionine-S-oxide reductase
  • cytosolic methionine-S-sulfoxide reductase
  • methionine sulfoxide reductase A
  • mitochondrial peptide methionine sulfoxide reductase
  • peptide Met(O) reductase
  • peptide met (O) reductase
  • peptide methionine sulfoxide reductase
  • peptide-methionine (S)-S-oxide reductase
  • protein-methionine-S-oxide reductase
Description
From NCBI Gene: This gene encodes a ubiquitous and highly conserved protein that carries out the enzymatic reduction of methionine sulfoxide to methionine. Human and animal studies have shown the highest levels of expression in kidney and nervous tissue. The protein functions in the repair of oxidatively damaged proteins to restore biological activity. Alternative splicing results in multiple transcript variants. [provided by RefSeq, May 2014]
Attributes
QA State
Under Review
Type
Gene
HGNC Name
MSRA
Certifications
  • None
QA State for Prostate
Under Review

 Non-Public Biomarker

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 Non-Public Biomarker

Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.

 Non-Public Biomarker

Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.

 Non-Public Biomarker

Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.

 Non-Public Biomarker

Organ-specific information for this biomarker is currently being annotated or is "under review". Logging in may give you privileges to view additional information. Contact the Informatics Center if you believe you should have access.